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Instead of treating symptoms at the surface, we uncover why theyre happening then use targeted testing, foundational correction, and supportive therapies to create predictable, measurable results

In this context, the exploration of ME/CFS pathogenesis has increasingly focused on the microbiome-gut-brain axis, revealing a complex interplay between intestinal dysbiosis and the multifaceted symptomatology of this debilitating condition [10]

doi:10.2337/db13-1093 Nishikawa T, Edelstein D, Du XL, Yamagishi S, Matsumura T, Kaneda Y, Yorek MA, Beebe D, Oates PJ, Hammes HP, Giardino I, Brownlee M (2000) Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

In turn, when the hypothalamus releases its GHRH, receptors in the pituitary gland nearby are stimulated to release HGH, or Human Growth Hormone